<html><head></head><body style="word-wrap: break-word; -webkit-nbsp-mode: space; -webkit-line-break: after-white-space; ">Hi, guys<div><br></div><div>I'd like to announce that GED journal club will be at 12pm on Wednesday (Sorry Jacob) and it will start next week.</div><div>I have already drafted the schedule of all speakers below. Please let me know if you cannot make it so I will switch you.</div><div><br></div><div>Likit 1/26</div><div>QP 2/2</div><div>Jaron 2/9</div><div>Neem 2/16</div><div>Adina 2/23</div><div>Jason 3/2</div><div>Elijah 3/16</div><div>Owen 3/23</div><div>Rose 3/30</div><div><br></div><div><br></div><div>***Remember: the article has to be sent out at least a week in advance or the journal club will be cancelled.***</div><div><br></div><div>When send out your selected paper via email, please follow this format if possible. Try not to attach a PDF file in the email.</div><div>The format is recommended by Titus.</div><div><br></div><div>For the first week, I will talk about this paper:</div><div><br></div><div><a href="http://www.nature.com/icb/journal/vaop/ncurrent/full/icb201083a.html">http://www.nature.com/icb/journal/vaop/ncurrent/full/icb201083a.html</a></div><div><br></div><div>Note or abstract:</div><div>MxA is an antiviral protein that inhibit the replication of many RNA viruses.</div><div>However, Taiwanese scientists have shown that HSV-1 virus can induce the expression of MxA isoform (varMxA) that facilitate its replication.</div><div>The antagonistic function of two isoforms of the same gene is very interesting. </div><div>This finding supports my hypothesis that alternative splicing of host mRNA can be induced by pathogens and it plays a crucial role in the immune response to viruses.</div><div><br></div><div>Cheers.</div></body></html>